E os átrios recebem a atenção que merecem na fibrilação atrial!

Figure 1 A, Note that the LA and posterior mitral annulus rest within the mouth of the LV inlet. The posterior annulus is related to the LA internally and to the crest of the LV inlet externally. The mitral leaflets normally rest in the plane of the mitral annulus. B, With atriogenic leaflet tethering, the posterior pole of the mitral annulus (P) becomes displaced by mounting the crest of the LV inlet. As a result, the ventricular surface of the PML becomes pressed against crest of the LV inlet and is no longer able to coapt effectively with the AML. At the same time, the plane of the mitral annulus (dashed line) becomes displaced superiorly (small arrows). This increases annulo‐papillary distance such that the mitral leaflets become tethered into the LV cavity. A–anterior pole of the mitral annulus

Mechanistic insights into atrial functional mitral regurgitation: Far more complicated than just left atrial remodeling

https://onlinelibrary.wiley.com/doi/full/10.1111/echo.14249

Recent studies lend credibility to the notion that lone atrial fibrillation (AF) can cause functional mitral regurgitation (MR), commonly referred to as atrial functional MR (AF‐MR). The conventional view holds that left atrial enlargement associated with AF causes annular dilatation which gradually moves the mitral valve leaflets apart resulting in inadequate coaptation and regurgitation. Recent findings, however, suggest that AF‐MR is not solely related to left atrial remodeling, but that important structural and functional abnormalities of the left ventricle also play a role in its pathogenesis.

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Estudos recentes dão credibilidade à noção de que a fibrilação atrial isolada (FA) pode causar insuficiência mitral funcional (RM), comumente referida como RM funcional atrial (AF ‐ MR). A visão convencional sustenta que o aumento do átrio esquerdo associado à FA causa dilatação anular que afasta gradualmente os folhetos da válvula mitral, resultando em coaptação e regurgitação inadequadas. Achados recentes, no entanto, sugerem que a FA ‐ RM não está relacionada apenas à remodelação do átrio esquerdo, mas que importantes anormalidades estruturais e funcionais do ventrículo esquerdo também desempenham um papel em sua patogênese.

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